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Krivic and colleagues reported promoted tendon-to-bone healing in a rat Achilles detachment model, and Staresinic and colleagues reported accelerated healing of transected rat Achilles tendon with tendocyte outgrowth in vitro

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ROS or lipid peroxidation products and consequently modified proteins may activate the ER stress-initiated apoptotic pathway, which initiator is caspase-2, with also increased levels in UV radiated keratinocytes [110]