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The sequential steps in the transition from postprandial to early and long-term fasting metabolism in adult individuals encompass a shift from glucose to lipid and ketone body metabolism, through consistent and complementary responses in the liver, skeletal muscle, and white adipose tissue (WAT), coordinated systemically by hormonal signaling

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3H), prenatal stress exposure upregulated genes associated with metabolite and energy production and transcription-factor binding, while downregulated genes were enriched for gliogenesis, cell adhesion, and integrin binding, suggesting suppression of glial and structural pathways