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The subjects here exhibited relatively little cognitive deficit or mitochondrial dysfunction in the hippocampus, and their cognitive function was comparable to that of the control group that did not receive isoflurane.8 Alzheimer’s disease Mitochondrial dysfunction appears to be a central event in the pathogenesis of Alzheimer’s disease, so the preservation of mitochondrial health may be one of the keys to disease prevention.9 To that end, a 2017 study points favorably to SS-31’s use for Alzheimer’s prevention

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Glutathione reductase then regenerates GSH using NADPH as a cofactor