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Therefore, we investigated the activation of FOXO3, which is associated with disruption of the BBB [29] and increased proapoptotic gene expression [58], as well as the activation of PI3K/Akt signaling, which is an upstream target of FOXO3 transcription [58, 59] and a cell survival pathway [60, 61]

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Oxidative phosphorylation is a metabolic vulnerability of endocrine therapy-tolerant persister cells in ER + breast cancer