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Traditionally, AD has been treated with drugs that relieve symptoms, such as cholinesterase inhibitors (donepezil, rivastigmine, and galantamine) and N-methyl-D-aspartate receptor (NMDAR) antagonists (memantine)

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[288] utilized type II collagens, HA, and chondroitin sulfate to construct an ideal microenvironment that preserved the normal chondrocytes phenotype

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