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This was illustrated by the decomposition of GSNO by γ-glutamyltranspeptidase (GGT) in the presence of trace metal ions [25]

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aeruginosa ΔgshA, ΔgshB and ΔgshAΔgshB double mutants defective in GSH synthesis were attenuated in a mouse model of acute pneumonia [27] yet the ΔgshA mutant was equally virulent as the wildtype in surgical wound, abscess and acute burn wound murine infection models [28]