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Although the exact mechanism of interstitial cystitis is unknown, there is thought to be a significant contribution from chronic inflammation, therefore BPC-157 was chosen in this study due to its anti-inflammatory properties and its role in restoring damaged epithelium as demonstrated in various animal models [22, 66, 67]

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Peripheral administration of an angiotensin II AT(1) receptor antagonist decreases the hypothalamic-pituitary-adrenal response to isolation Stress