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GHRs are involved in the catabolism of chlorinated quinones and in lignin degradation through the deglutathionylation of metabolic intermediates (Reddy and Gold, 2001

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Engagement of interleukin-17A (IL-17A) with the interleukin-17 receptor A/C (IL-17RA/RC) complex may contribute to activation of the transforming growth factor-β–activated kinase 1 (TAK1)–IκB kinase (IKK)–nuclear factor κ-light-chain-enhancer of activated B cells (NF-κB) signaling axis, thereby amplifying pro-inflammatory mediators—including IL-6, TNF-α, and interleukin-1β (IL-1β)—and promoting a self-reinforcing inflammatory cascade (52)