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This prompts us to consider that besides environmental serine/glycine concentration, other variables probably also are important to induce the NKX2–1 dependent transcriptional programme, indicating that our observations might be an underestimation of the human situation, especially when leukaemic stem cells encounter hypoxic niches in the bone marrow and subsequent limited availability of many nutrients

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encoded by the NFE2L2 ) plays a key role in regulating cellular redox homeostasis and the protective antioxidant detoxification mechanisms in mammals [1]