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At the same time, it can reverse the decreased expression of BDNF in the prefrontal cortex and hippocampus, and then regulate the BDNF-TrkB and downstream AKT-mTOR and ERK signaling pathways, promote the increase of synaptic protein expression levels, thus protecting neurons, enhancing synaptic plasticity, improving the depression-like behaviors of model animals, and playing an effective antidepressant role (Fig

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Exp Hematol (2000) 28(3):294–304.10.1016/S0301-472X(99)00154-X 21 TianSSTapleyPSincichCSteinRBRosenJLambP