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More broadly, colonizing cells flex iron and membrane-lipid economies to avoid lethal peroxidation in the lung microenvironment: autophagy–iron crosstalk and transferrin receptor routing shape labile iron pools and lipid ROS thresholds during early outgrowth, as seen when restraining autophagic TFRC turnover triggers iron accumulation, lipid peroxidation, and impaired metastatic behavior in colorectal models that otherwise seed lung [133]

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J., Rego I., Ruiz-Romero C