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Knocking down ETT causes increased levels of oxidative damage, as reflected by increase in protein carbonylation, lipid peroxidation, and DNA damage (102)
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Discussion Non-alcoholic fatty liver disease is a multifactorial disease with a complex pathogenesis including liver alterations with increased lipid accumulation, with or without inflammation, and without other causes of liver disease ( An animal model of HFD-induced NAFLD was established to outline the etiology, histopathology, and disease progression ( de novo lipogenesis, fatty acid uptake, fatty acid oxidation, and TG output ( Non-alcoholic fatty liver disease is closely related to abnormal metabolic profiles in vivo
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