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This theory also proposes that malfunction of an as yet undiscovered lactate shuttle within the NMJ may be a pathological feature of ALS, suggesting that supporting mitochondrial function may optimize lactate metabolism and combat the toxicity caused by accumulation of excess lactate

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In experimental models of pulmonary fibrosis, NAC has been shown to alleviate disease progression by scavenging ROS, mitigating oxidative damage, and protecting alveolar epithelial cells, with its therapeutic potential validated by multiple studies (Mokra et al., 2023)