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Importantly, in vivo MARCHF6 deletion led to severe hepatic injury and perinatal lethality in mice, which were partly rescued by lipophilic antioxidants, highlighting the therapeutic relevance of the NADPH–MARCHF6 axis in liver physiology and ferroptosis regulation [27], [35] (Fig

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doi: 10.1016/j.lfs.2023.122370 411 HuangCGuoYLiTSunGYangJWangYet al