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It has also been shown that coronavirus infection can inhibit the activity of numerous karyopherin-dependent host transcription factors including the vitamin D receptor (VDR) and hypoxia-inducible factor α2 [HIFα2]-Epas, p53 in human lung cells [109]

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Within the complex disease context, fundamental questions remain regarding the specificity, detectability, and spatiotemporal dynamics of ferroptosis, as well as its interactions with classical pathological pathways such as Aβ, tau, α-synuclein, and mutant huntingtin protein