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The aim of the present study is that the free radicals may react with biomolecules of the cells and play an important role in the development of chronic disease conditions in the humans

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Prior literature has shown that NNMT silencing in mouse adipocytes significantly upregulated intracellular SAM levels and promoted the expression and activity of ornithine decarboxylase (ODC) and SSAT by increasing the methylation level of histone H3K4, thereby promoting polyamine metabolism and energy consumption [44]