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Acute intermittent hypoxia (AIH) is one of the most promising approaches to improve recovery after spinal cord injury and other diseases with motoneurons alterations due to its effects on neuronal plasticity through increasing VEGF, brain-derived neurotrophic factor (BDNF), and phosphorylated and non-phosphorylated forms of the BDNF receptor tropomyosin-related kinase B (TrkB) (Hassan et al., 2018