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Excess ROS then activates the NLRP3 inflammasome in microglia, promoting maturation and release of IL-1β ( 4.3 GSH metabolism: a central hub in mid-late SAE The hippocampus, with its high metabolic demands and low antioxidant capacity, is uniquely vulnerable to septic injury ( A Stat1-Gpx3 hub was identified, where TNF-α/IL-1β-induced Stat1 activation suppresses Nrf2 (confirmed by reduced hippocampal Nrf2)

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Our own preclinical work has suggested that behavioral and cognitive deficits are attributed to three brain regions: hippocampus, amygdala, and prefrontal cortex (PFC) (Masood et al., 2008