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The author suggested that the reduction of cellular tHcy was the cause, rather than the renal clearance, which is influenced by genotype

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6 Future directions and clinical perspectives Despite significant advances in delineating the role of eryptosis in the pathogenesis of renal anemia—including its regulation by uremic toxins (e.g., indoxyl sulfate, acrolein, oxidative stress, and inflammatory mediators—critical gaps remain in translating mechanistic insights into clinical practice, presenting several promising avenues for future research

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