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While oxidative stress contributes to cancer progression, understanding the complex interplay between obesity-induced metabolic changes and redox signaling in triple negative breast cancer (TNBC) presents a unique opportunity to develop targeted therapeutic strategies for this historically treatment-resistant disease.In this dissertation, we aimed to understand how obesity and TNBC, both as metabolic diseases, affect lipid-specific oxidative stress in the context of tumor metabolism and response to current front-line therapies

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62 Knocking out IL-1 receptor 1 (IL-1R1) or pharmacologic inhibition of IL-1 signaling in older mice reversed the myeloid-biased hematopoietic output